Mesenteric ischemia? (CTA)
Pain out of proportion to examination, often with AF or a low-flow state; every hour of delay costs bowel.
Answer the surgeon
Emergent revascularization plus resection of non-viable bowel vs anticoagulation (venous) vs treating the low-flow state (NOMI).
Your answer must state:
- Mechanism: SMA embolus (abrupt mid-SMA cutoff, no collaterals, AF) vs in-situ thrombosis (ostial, calcified plaque, collaterals) vs SMV thrombosis vs NOMI (diffuse narrowing, vasopressors)
- Bowel viability: wall enhancement present or absent
- Advanced bowel-injury signs: interpret pneumatosis and portomesenteric venous gas in context; identify frank perforation
Key and direct findings
Supportive signs & checks
Wet-read snippets
Fill the blanks, then copy into your preliminary report.
Positive
Acute mesenteric ischemia: ___ (SMA embolic occlusion ___ cm from origin / ostial SMA thrombosis / SMV thrombosis) with ___ bowel-wall enhancement of ___. Pneumatosis/portal venous gas: ___.
Negative
Patent SMA, SMV and celiac axis with normal bowel-wall enhancement — no CTA evidence of mesenteric ischemia.
Sources
- WSES guidelines for acute mesenteric ischemia (2022 update) — Bala M et al. World J Emerg Surg (2022)
- Diagnostic accuracy of multidetector CTA in acute mesenteric ischemia — Menke J. Radiology (2010)
Content review: Content pending clinical review · 2026-07-15
Educational reference for trained clinicians. Verify every result against the cited original guideline before use. Not a substitute for clinical judgment or medical advice.